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Genetic knock out of Cygb promotes hepatic fibrosis and the development of HCC, accompanied by increased markers of oxidative stress under the administration of diethylnitrosamine [28] or a choline-deficient amino acid-defined diet [29]

These metabolic processes are vital for sustaining the integrity of the nervous system, as the energy produced is critical for the functioning of nerve cells

However, folate supplementation can mask these haematological changes whilst neurological damage continues, highlighting the importance of checking B12 status before commencing folate therapy